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Breaks in sitting may augment reductions in postprandial glucose and insulin responses via elevations in energy expenditure

机译：坐位休息可以通过能量消耗的增加来增加餐后葡萄糖和胰岛素反应的减少

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R.N. Larsen; P. Dempsey; F. Dillon; M. Grace; B.A. Kingwell; N. Owen; D.W. Dunstan;
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年度 2017
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1. The effects of prolonged sitting, prolonged standing, and activity breaks on vascular function, and postprandial glucose and insulin responses: A randomised crossover trial [J] . Meredith C. Peddie, Chris Kessell, Tom Bergen, PLoS One . 2021,第1期

机译：长时间坐，长时间的身份和活动突破对血管功能的影响，以及后葡萄糖和胰岛素反应：随机交叉试验
2. Breaking up prolonged sitting reduces postprandial glucose and insulin responses. [J] . David W Dunstan, Bronwyn A Kingwell, Robyn Larsen, Diabetes care . 2012,第5期

机译：延长长时间坐着可以减少餐后血糖和胰岛素反应。
3. Breaking up of prolonged sitting over three days sustains, but does not enhance, lowering of postprandial plasma glucose and insulin in overweight and obese adults [J] . Larsen Robyn N., Kingwell Bronwyn A., Robinson Carol, Clinical Science . 2015,第1a2期

机译：超过三天的长时间坐着休息可以维持，但不能增强超重和肥胖成年人的餐后血浆葡萄糖和胰岛素的降低
4. Mathematical model of postprandial glucose-insulin metabolism in insulin dependent diabetics with subcutaneous and intravenous insulin administration routes [C] . Yamamoto Cecilia, Furutani Eiko, Sumi Shoichiro SICE Annual Conference 2011 : Final program and abstracts . 2011

机译：胰岛素依赖型糖尿病患者皮下和静脉内胰岛素给药途径的餐后葡萄糖-胰岛素代谢数学模型
5. The ability of the SenseWearRTM Armband to assess a change in energy expenditure in children while sitting and standing. [D] . Mancuso, Alexandrea Rose. 2010

机译：SenseWearRTM臂环评估坐着和站着儿童的能量消耗变化的能力。
6. Breaking Up Prolonged Sitting Reduces Postprandial Glucose and Insulin Responses [O] . David W. Dunstan, Bronwyn A. Kingwell, Robyn Larsen, 2012

机译：长时间休息可以减少餐后血糖和胰岛素反应
7. The effects of prolonged sitting, prolonged standing, and activity breaks on vascular function, and postprandial glucose and insulin responses: A randomised crossover trial [O] . Meredith C. Peddie, Chris Kessell, Tom Bergen, 2021

机译：长期坐，长时间的身份和活性突破对血管功能的影响，以及后葡萄糖和胰岛素反应：随机交叉试验

Breaks in sitting may augment reductions in postprandial glucose and insulin responses via elevations in energy expenditure

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